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miR-29b might disrupt glucose metabolism and suppress epithelial ovarian most cancers growth by decreasing AKT2-AKT3 and PKM2 degreesKDM8, a histone lysine demethylase, binds to PKM2, blocking PKM2 tetramer development and advertising and marketing its nuclear translocation. Nuclear PKM2 serves as being a protein kinase to phosphorylate histone H3